ICD-10: G46.5

Pure motor lacunar syndrome

Additional Information

Approximate Synonyms

Pure motor lacunar syndrome, classified under ICD-10 code G46.5, is a specific type of stroke that primarily affects motor function due to small vessel disease. Understanding its alternative names and related terms can enhance clarity in medical communication and documentation. Below are some of the alternative names and related terms associated with this condition.

Alternative Names

  1. Lacunar Stroke: This term broadly refers to strokes caused by occlusion of small penetrating arteries that supply deep structures of the brain, including the basal ganglia and thalamus. Pure motor lacunar syndrome is a subtype of lacunar stroke.

  2. Pure Motor Hemiparesis: This term emphasizes the motor deficits experienced by patients, typically manifesting as weakness on one side of the body without sensory loss.

  3. Lacunar Infarct: While this term refers to the actual infarct (tissue death) caused by the blockage of small vessels, it is often used interchangeably with pure motor lacunar syndrome when discussing the clinical presentation.

  4. Small Vessel Disease: This term describes the underlying pathology that leads to pure motor lacunar syndrome, highlighting the role of small vessel occlusion in the brain.

  1. Cerebrovascular Accident (CVA): A general term for any disruption of blood flow to the brain, which includes strokes of various types, including lacunar strokes.

  2. Ischemic Stroke: This term refers to strokes caused by a blockage in a blood vessel, which encompasses lacunar strokes.

  3. Motor Function Impairment: A broader term that describes the loss of motor skills, which is a primary symptom of pure motor lacunar syndrome.

  4. Neurological Deficits: This term encompasses a range of symptoms that may arise from brain injuries, including those seen in pure motor lacunar syndrome.

  5. Thalamic Stroke: While not exclusively synonymous, thalamic strokes can lead to similar motor deficits and may be discussed in the context of pure motor lacunar syndrome.

Conclusion

Understanding the alternative names and related terms for ICD-10 code G46.5: Pure motor lacunar syndrome is crucial for accurate diagnosis, treatment, and communication among healthcare professionals. These terms reflect the condition's clinical features and underlying pathology, facilitating better patient care and research discussions. If you have further questions or need more specific information, feel free to ask!

Description

Pure motor lacunar syndrome, classified under ICD-10 code G46.5, is a specific type of stroke that primarily affects motor function. This condition is characterized by the presence of motor deficits without accompanying sensory or cognitive impairments, which distinguishes it from other types of strokes.

Clinical Description

Definition

Pure motor lacunar syndrome is a neurological condition resulting from small, deep infarcts in the brain, typically caused by chronic hypertension or lipohyalinosis. These small strokes occur in the deep structures of the brain, such as the basal ganglia, thalamus, or internal capsule, leading to isolated motor symptoms.

Symptoms

Patients with pure motor lacunar syndrome typically present with:
- Weakness: This is often unilateral and can affect the arm, leg, or both, depending on the location of the infarct.
- Spasticity: Increased muscle tone may be observed, leading to stiffness and difficulty in movement.
- Reflex Changes: Hyperreflexia may be present, indicating an upper motor neuron lesion.

Importantly, sensory functions remain intact, and there are no significant cognitive deficits associated with this syndrome, which helps differentiate it from other stroke types that may involve both motor and sensory pathways[1][2].

Pathophysiology

The underlying mechanism of pure motor lacunar syndrome involves the occlusion of small penetrating arteries that supply deep structures of the brain. This occlusion can be due to:
- Atherosclerosis: Hardening of the arteries leading to reduced blood flow.
- Lipohyalinosis: Degenerative changes in the small vessels, often associated with chronic hypertension and diabetes.

These small vessel diseases result in localized ischemia, leading to the characteristic motor deficits seen in patients[3][4].

Diagnosis

Diagnosis of pure motor lacunar syndrome typically involves:
- Clinical Assessment: A thorough neurological examination to assess motor function and rule out other conditions.
- Imaging Studies: MRI or CT scans are used to identify the presence of lacunar infarcts in the brain. MRI is particularly useful for visualizing small lesions that may not be evident on CT scans[5].

Treatment

Management of pure motor lacunar syndrome focuses on:
- Acute Management: Immediate treatment may include antiplatelet therapy to prevent further strokes.
- Long-term Management: Control of risk factors such as hypertension, diabetes, and hyperlipidemia is crucial. Rehabilitation therapies, including physical and occupational therapy, are often recommended to improve motor function and quality of life[6][7].

Prognosis

The prognosis for patients with pure motor lacunar syndrome can vary. Many patients experience significant recovery, especially with early intervention and rehabilitation. However, some may have persistent motor deficits, which can impact daily activities and overall quality of life[8].

In summary, pure motor lacunar syndrome (ICD-10 code G46.5) is a distinct clinical entity characterized by isolated motor deficits due to small, deep brain infarcts. Understanding its clinical features, pathophysiology, and management strategies is essential for effective treatment and rehabilitation.


References

  1. ICD-10-CM Code for Pure motor lacunar syndrome G46.5.
  2. ICD-10 code: G46.5 Pure motor lacunar syndrome.
  3. ICD-10 code G46 for Vascular syndromes of brain.
  4. ICD-10-CM Diagnosis Code G46.5 - Pure motor lacunar syndrome.
  5. CodeMapĀ® ICD-10 (CM) Report.
  6. Billing and Coding: Monitored Anesthesia Care.
  7. Identifying genetic variants associated with the ICD10.
  8. ICD-10-CM TABULAR LIST of DISEASES and INJURIES.

Clinical Information

Pure motor lacunar syndrome, classified under ICD-10 code G46.5, is a specific type of stroke that results from small vessel disease, leading to lacunar infarcts in the brain. This condition is characterized by a distinct clinical presentation, including various signs and symptoms that can significantly impact a patient's motor function.

Clinical Presentation

Definition and Etiology

Pure motor lacunar syndrome is primarily caused by the occlusion of small penetrating arteries that supply deep structures of the brain, such as the basal ganglia, internal capsule, and thalamus. This syndrome is often associated with chronic conditions like hypertension and diabetes, which contribute to small vessel disease and subsequent lacunar infarcts[1][2].

Signs and Symptoms

Patients with pure motor lacunar syndrome typically present with the following signs and symptoms:

  • Hemiparesis: The most prominent feature is weakness on one side of the body, often affecting the arm and leg more than the face. This weakness is usually contralateral to the side of the brain where the infarct occurred[1].
  • Facial Weakness: While the face may be less affected than the limbs, some patients may exhibit mild facial droop on the affected side[2].
  • Spasticity: Increased muscle tone and spasticity may develop in the affected limbs, leading to stiffness and difficulty with movement[1].
  • Reflex Changes: Hyperreflexia (increased reflexes) may be observed, indicating upper motor neuron involvement[2].
  • Sensory Function: Unlike other types of strokes, pure motor lacunar syndrome typically does not present with significant sensory deficits, which helps differentiate it from other stroke syndromes[1][2].

Patient Characteristics

Patients who develop pure motor lacunar syndrome often share certain characteristics:

  • Age: This syndrome is more common in older adults, particularly those over the age of 60[1].
  • Comorbidities: A history of hypertension, diabetes mellitus, and hyperlipidemia is frequently noted, as these conditions contribute to the underlying small vessel disease[2].
  • Lifestyle Factors: Patients may have lifestyle risk factors such as smoking and sedentary behavior, which exacerbate vascular health issues[1].
  • Gender: There may be a slight male predominance in the incidence of lacunar strokes, although both genders are affected[2].

Conclusion

Pure motor lacunar syndrome, represented by ICD-10 code G46.5, is characterized by specific motor deficits resulting from small vessel occlusion in the brain. The clinical presentation typically includes hemiparesis, spasticity, and reflex changes, with minimal sensory involvement. Understanding the signs, symptoms, and patient characteristics associated with this syndrome is crucial for timely diagnosis and management, particularly in populations at risk due to age and comorbid conditions. Early intervention can significantly improve outcomes and quality of life for affected individuals.

Diagnostic Criteria

Pure motor lacunar syndrome, classified under ICD-10 code G46.5, is a specific type of stroke that results from small vessel disease, leading to motor function impairment without sensory loss. Understanding the diagnostic criteria for this condition is crucial for accurate coding and treatment. Below, we explore the key criteria and considerations involved in diagnosing pure motor lacunar syndrome.

Diagnostic Criteria for Pure Motor Lacunar Syndrome

Clinical Presentation

  1. Motor Deficits: The hallmark of pure motor lacunar syndrome is the presence of weakness in the face, arm, or leg, typically on one side of the body. This weakness is often described as hemiparesis and can vary in severity.
  2. Absence of Sensory Loss: Unlike other types of strokes, pure motor lacunar syndrome does not present with sensory deficits. Patients may exhibit normal sensory function despite significant motor impairment.
  3. Acute Onset: Symptoms usually appear suddenly, consistent with the acute nature of stroke events. The rapid onset of motor weakness is a critical factor in diagnosis.

Neurological Examination

  1. Focal Neurological Signs: A thorough neurological examination is essential. Physicians look for specific focal signs, such as:
    - Weakness in the upper or lower extremities.
    - Possible facial droop, particularly affecting the lower part of the face.
  2. Reflexes: Increased deep tendon reflexes may be observed, indicating upper motor neuron involvement.

Imaging Studies

  1. MRI or CT Scans: Neuroimaging plays a vital role in confirming the diagnosis. Imaging studies may reveal:
    - Small, deep infarcts in the basal ganglia, internal capsule, or thalamus, which are characteristic of lacunar strokes.
    - Absence of significant cortical involvement, which helps differentiate pure motor lacunar syndrome from other stroke types.

Exclusion of Other Conditions

  1. Differential Diagnosis: It is essential to rule out other potential causes of motor weakness, such as:
    - Other types of strokes (e.g., hemorrhagic or cortical strokes).
    - Neurological conditions like multiple sclerosis or amyotrophic lateral sclerosis (ALS).
    - Peripheral nerve injuries or myopathies.

Risk Factors

  1. Underlying Conditions: The presence of risk factors such as hypertension, diabetes, and hyperlipidemia is often associated with small vessel disease and can support the diagnosis of pure motor lacunar syndrome.

Conclusion

Diagnosing pure motor lacunar syndrome (ICD-10 code G46.5) involves a combination of clinical assessment, neurological examination, imaging studies, and exclusion of other conditions. The key features include acute onset of unilateral motor weakness without sensory loss, supported by neuroimaging findings of small deep infarcts. Accurate diagnosis is essential for appropriate management and coding, ensuring that patients receive the necessary care for their condition.

Treatment Guidelines

Pure motor lacunar syndrome, classified under ICD-10 code G46.5, is a type of stroke that results from small vessel disease, leading to motor deficits without sensory loss. This condition is characterized by weakness in the face, arm, or leg, typically on one side of the body, and is often associated with hypertension, diabetes, and other vascular risk factors. Understanding the standard treatment approaches for this syndrome is crucial for effective management and rehabilitation.

Overview of Pure Motor Lacunar Syndrome

Pure motor lacunar syndrome occurs due to the occlusion of small penetrating arteries that supply deep structures of the brain, such as the basal ganglia, internal capsule, or thalamus. The clinical presentation usually includes:

  • Hemiparesis (weakness on one side of the body)
  • Dysarthria (difficulty speaking)
  • No sensory deficits, distinguishing it from other types of strokes[1].

Standard Treatment Approaches

1. Acute Management

In the acute phase of pure motor lacunar syndrome, the primary goal is to stabilize the patient and prevent further neurological damage. Key interventions include:

  • Antiplatelet Therapy: Aspirin is commonly prescribed to reduce the risk of recurrent strokes. In some cases, clopidogrel may be used, especially if the patient has a history of cardiovascular events[2].
  • Management of Risk Factors: Immediate attention to controlling blood pressure, blood glucose levels, and cholesterol is critical. This may involve the use of antihypertensive medications, insulin or oral hypoglycemics for diabetes, and statins for hyperlipidemia[3].

2. Rehabilitation

Rehabilitation plays a vital role in recovery from pure motor lacunar syndrome. The focus is on restoring function and improving quality of life:

  • Physical Therapy: Tailored exercises to improve strength, coordination, and mobility are essential. Therapists may use techniques such as task-specific training and gait training to enhance recovery[4].
  • Occupational Therapy: This helps patients regain independence in daily activities. Occupational therapists work on fine motor skills and adaptive strategies to manage daily tasks[5].
  • Speech Therapy: If dysarthria is present, speech therapy can assist in improving communication skills and swallowing function[6].

3. Long-term Management

Long-term management focuses on preventing recurrence and managing chronic conditions:

  • Lifestyle Modifications: Encouraging a heart-healthy diet, regular physical activity, smoking cessation, and weight management can significantly reduce the risk of future strokes[7].
  • Regular Monitoring: Patients should have regular follow-ups to monitor blood pressure, glucose levels, and overall cardiovascular health. Adjustments to medications may be necessary based on these evaluations[8].

4. Patient Education and Support

Educating patients and their families about the condition, treatment options, and lifestyle changes is crucial. Support groups and counseling can also provide emotional support and resources for coping with the challenges of recovery[9].

Conclusion

The management of pure motor lacunar syndrome involves a comprehensive approach that includes acute treatment, rehabilitation, long-term risk factor management, and patient education. By addressing both the immediate and ongoing needs of patients, healthcare providers can significantly improve outcomes and enhance the quality of life for those affected by this condition. Regular follow-up and adherence to treatment plans are essential for preventing recurrence and promoting recovery.

Related Information

Approximate Synonyms

  • Lacunar Stroke
  • Pure Motor Hemiparesis
  • Lacunar Infarct
  • Small Vessel Disease
  • Cerebrovascular Accident (CVA)
  • Ischemic Stroke
  • Motor Function Impairment
  • Neurological Deficits
  • Thalamic Stroke

Description

  • Specific type of stroke affecting motor function
  • Motor deficits without sensory or cognitive impairments
  • Caused by small, deep infarcts in brain structures
  • Typically caused by chronic hypertension or lipohyalinosis
  • Patients present with weakness and spasticity
  • Sensory functions remain intact with no significant cognitive deficits

Clinical Information

  • Caused by small vessel disease
  • Results from lacunar infarcts
  • Typically affects older adults
  • Common in patients with hypertension
  • Diabetes mellitus is also common
  • Hemiparesis is the most prominent feature
  • Facial weakness may be present
  • Spasticity and reflex changes occur
  • Sensory function is typically preserved

Diagnostic Criteria

  • Motor deficits in face, arm, or leg
  • Absence of sensory loss
  • Acute onset of motor weakness
  • Focal neurological signs
  • Increased deep tendon reflexes
  • Small, deep infarcts on MRI/CT scans
  • Exclusion of other conditions
  • Presence of risk factors (hypertension, diabetes, etc.)

Treatment Guidelines

  • Aspirin prescribed for antiplatelet therapy
  • Control blood pressure levels immediately
  • Manage diabetes through insulin or oral hypoglycemics
  • Use statins for hyperlipidemia treatment
  • Physical therapy focuses on strength and mobility
  • Occupational therapy promotes independence in daily tasks
  • Speech therapy addresses dysarthria and swallowing issues

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