ICD-10: I63.339

Cerebral infarction due to thrombosis of unspecified posterior cerebral artery

Additional Information

Description

Cerebral infarction, particularly due to thrombosis of the posterior cerebral artery, is a significant medical condition that can lead to various neurological deficits. The ICD-10 code I63.339 specifically refers to a cerebral infarction caused by thrombosis in an unspecified posterior cerebral artery. Below is a detailed clinical description and relevant information regarding this condition.

Clinical Description

Definition

Cerebral infarction occurs when blood flow to a part of the brain is obstructed, leading to tissue death (infarction) due to a lack of oxygen and nutrients. The posterior cerebral artery (PCA) supplies blood to the occipital lobe, parts of the temporal lobe, and the thalamus. Thrombosis in this artery can result in significant neurological impairments, particularly affecting vision and sensory processing.

Etiology

The primary cause of cerebral infarction due to thrombosis is the formation of a blood clot within the artery, which can be precipitated by various factors, including:
- Atherosclerosis: The buildup of fatty deposits in the arteries.
- Cardiac embolism: Clots that form in the heart and travel to the brain.
- Hypercoagulable states: Conditions that increase the tendency for blood to clot.

Symptoms

Patients with cerebral infarction due to thrombosis of the PCA may present with a range of symptoms, including:
- Visual disturbances: Such as homonymous hemianopia (loss of half of the visual field).
- Sensory deficits: Including numbness or tingling in the limbs.
- Cognitive impairments: Depending on the extent of the infarction and areas affected.
- Difficulty with balance and coordination.

Diagnosis

Diagnosis typically involves:
- Clinical evaluation: Assessing symptoms and medical history.
- Imaging studies: CT or MRI scans are crucial for visualizing the infarction and determining its location and extent.
- Vascular studies: Such as Doppler ultrasound or angiography, to assess blood flow and identify thrombosis.

Treatment

Management of cerebral infarction due to thrombosis may include:
- Acute interventions: Such as thrombolysis (clot-busting drugs) or thrombectomy (surgical removal of the clot) if the patient presents within a certain time frame.
- Antiplatelet therapy: To prevent further clot formation.
- Rehabilitation: Physical, occupational, and speech therapy to aid recovery and improve function.

Coding Details

ICD-10 Code I63.339

  • Code Description: Cerebral infarction due to thrombosis of unspecified posterior cerebral artery.
  • Classification: This code falls under the category of cerebrovascular diseases, specifically ischemic strokes due to thrombosis.
  • Use in Billing: Accurate coding is essential for proper billing and reimbursement in healthcare settings, ensuring that the specific nature of the infarction is documented for treatment and management purposes.

Importance of Specificity

The designation of "unspecified" in the code indicates that while the infarction is confirmed to be due to thrombosis in the PCA, the exact artery affected is not specified. This can impact treatment decisions and the understanding of the patient's condition, highlighting the importance of thorough diagnostic imaging and clinical assessment.

Conclusion

ICD-10 code I63.339 represents a critical aspect of cerebrovascular disease management, specifically addressing cerebral infarction due to thrombosis in the posterior cerebral artery. Understanding the clinical implications, symptoms, and treatment options associated with this condition is vital for healthcare providers in delivering effective care and improving patient outcomes. Accurate coding and documentation are essential for ensuring appropriate treatment and reimbursement in clinical practice.

Clinical Information

Cerebral infarction due to thrombosis of the unspecified posterior cerebral artery, classified under ICD-10 code I63.339, is a significant medical condition that requires a comprehensive understanding of its clinical presentation, signs, symptoms, and patient characteristics. Below, we delve into these aspects to provide a detailed overview.

Clinical Presentation

Cerebral infarction, commonly referred to as a stroke, occurs when blood flow to a part of the brain is interrupted, leading to tissue damage. In the case of I63.339, the infarction is specifically due to thrombosis in the posterior cerebral artery (PCA), which supplies blood to the occipital lobe and parts of the temporal lobe. The clinical presentation can vary widely depending on the extent of the infarction and the specific areas of the brain affected.

Signs and Symptoms

The signs and symptoms of a cerebral infarction due to thrombosis of the PCA may include:

  • Visual Disturbances: Patients may experience visual field deficits, such as homonymous hemianopia, where vision is lost in the same field of both eyes. This occurs due to the involvement of the occipital lobe, which is responsible for processing visual information[1].

  • Cognitive Impairments: Depending on the extent of the infarction, cognitive functions may be affected, leading to difficulties in memory, attention, and problem-solving abilities[1].

  • Sensory Loss: Patients may report numbness or weakness on one side of the body, particularly affecting the contralateral side to the infarction[1].

  • Motor Deficits: Weakness or paralysis (hemiparesis or hemiplegia) may occur, particularly affecting the arm and leg on the opposite side of the body from the infarction[1].

  • Speech Difficulties: Aphasia or difficulty in speech production may arise if the infarction affects areas involved in language processing[1].

  • Other Neurological Signs: Depending on the specific areas affected, patients may also present with ataxia, dysphagia, or altered consciousness[1].

Patient Characteristics

Understanding the demographic and clinical characteristics of patients who may experience a cerebral infarction due to thrombosis of the PCA is crucial for effective management and prevention strategies.

Demographics

  • Age: The risk of stroke increases with age, particularly in individuals over 55 years old. However, younger patients can also be affected, especially if they have underlying risk factors[1].

  • Gender: While both genders are at risk, some studies suggest that men may have a higher incidence of stroke at younger ages compared to women[1].

Risk Factors

Several risk factors are associated with an increased likelihood of developing a cerebral infarction, including:

  • Hypertension: High blood pressure is a significant risk factor for stroke, contributing to vascular damage and thrombosis[1].

  • Diabetes Mellitus: Diabetes can lead to vascular complications, increasing the risk of thrombotic events[1].

  • Hyperlipidemia: Elevated cholesterol levels can contribute to atherosclerosis, which may lead to thrombosis in cerebral arteries[1].

  • Smoking: Tobacco use is a well-known risk factor for stroke, as it contributes to vascular damage and increases clot formation[1].

  • Cardiac Conditions: Atrial fibrillation and other heart diseases can lead to embolic strokes, but they may also contribute to thrombotic strokes in certain contexts[1].

Clinical Characteristics

  • Comorbidities: Patients with a history of cardiovascular diseases, such as coronary artery disease or peripheral artery disease, are at higher risk for cerebral infarction[1].

  • Lifestyle Factors: Sedentary lifestyle, poor diet, and obesity are additional factors that can increase stroke risk[1].

Conclusion

Cerebral infarction due to thrombosis of the unspecified posterior cerebral artery (ICD-10 code I63.339) presents with a range of neurological symptoms primarily affecting vision, cognition, and motor function. Understanding the clinical presentation, signs, symptoms, and patient characteristics is essential for timely diagnosis and intervention. Identifying and managing risk factors can significantly reduce the incidence of such strokes, highlighting the importance of preventive healthcare measures. For healthcare providers, recognizing these aspects can lead to improved patient outcomes and better management strategies for those at risk.

Approximate Synonyms

ICD-10 code I63.339 refers specifically to a cerebral infarction caused by thrombosis in an unspecified posterior cerebral artery. Understanding alternative names and related terms for this diagnosis can enhance clarity in medical documentation and communication. Below are some relevant terms and synonyms associated with this condition.

Alternative Names

  1. Cerebral Infarction: This is a broader term that encompasses any type of stroke caused by a blockage in the blood supply to the brain, including those due to thrombosis.

  2. Ischemic Stroke: This term is often used interchangeably with cerebral infarction, specifically referring to strokes caused by a lack of blood flow due to a blockage.

  3. Thrombotic Stroke: This term highlights the specific mechanism of the stroke, indicating that it is caused by a thrombus (blood clot) obstructing a blood vessel.

  4. Posterior Cerebral Artery Infarction: While I63.339 specifies "unspecified," this term can be used when discussing infarctions specifically in the posterior cerebral artery region.

  1. Cerebral Thrombosis: This term refers to the formation of a blood clot within a blood vessel in the brain, which can lead to a cerebral infarction.

  2. Stroke: A general term that encompasses both ischemic and hemorrhagic strokes, but in the context of I63.339, it specifically refers to ischemic strokes.

  3. Neurological Deficit: This term describes the loss of normal function in the brain due to the infarction, which can manifest as various neurological symptoms.

  4. Transient Ischemic Attack (TIA): Although not the same as a cerebral infarction, a TIA is often related and can precede a full-blown stroke, indicating temporary blood flow disruption.

  5. Vascular Occlusion: This term refers to the blockage of a blood vessel, which is the underlying cause of the infarction.

  6. Cerebrovascular Accident (CVA): This is a medical term for a stroke, which can be ischemic (like I63.339) or hemorrhagic.

Conclusion

Understanding the alternative names and related terms for ICD-10 code I63.339 is crucial for healthcare professionals involved in diagnosis, treatment, and documentation. These terms not only facilitate clearer communication among medical staff but also enhance patient understanding of their condition. If you need further details or specific applications of these terms in clinical practice, feel free to ask!

Diagnostic Criteria

The ICD-10 code I63.339 refers to a specific type of cerebral infarction, namely, a cerebral infarction due to thrombosis of an unspecified posterior cerebral artery. Understanding the criteria for diagnosing this condition involves a combination of clinical evaluation, imaging studies, and specific diagnostic criteria.

Clinical Criteria for Diagnosis

  1. Symptoms and Clinical Presentation:
    - Patients may present with sudden onset of neurological deficits, which can include visual disturbances, sensory loss, or motor weakness, depending on the area of the brain affected by the infarction.
    - Common symptoms associated with posterior cerebral artery infarctions include homonymous hemianopia (loss of half of the visual field), memory deficits, and difficulties with spatial awareness[1].

  2. Neurological Examination:
    - A thorough neurological examination is essential to assess the extent of deficits. This may include evaluating cranial nerve function, motor strength, sensory function, and coordination[1].

Diagnostic Imaging

  1. CT or MRI Scans:
    - Computed Tomography (CT): A non-contrast CT scan is often the first imaging modality used to rule out hemorrhagic stroke. It can show early signs of ischemia, such as loss of gray-white matter differentiation or subtle hypodensities in the affected area.
    - Magnetic Resonance Imaging (MRI): MRI is more sensitive in detecting acute cerebral infarctions. Diffusion-weighted imaging (DWI) can identify areas of restricted diffusion, indicating recent ischemia[2].

  2. Angiography:
    - In some cases, cerebral angiography may be performed to visualize the blood vessels and confirm the presence of thrombosis in the posterior cerebral artery. This can help differentiate between embolic and thrombotic events[2].

Laboratory Tests

  1. Blood Tests:
    - Routine blood tests may be conducted to assess for risk factors such as hyperlipidemia, diabetes, and coagulation disorders. These tests can help identify underlying conditions that may contribute to thrombosis[1].

  2. Cardiac Evaluation:
    - An echocardiogram may be performed to evaluate for potential cardiac sources of emboli, especially if the infarction is suspected to be embolic rather than purely thrombotic[2].

Conclusion

The diagnosis of cerebral infarction due to thrombosis of the posterior cerebral artery (ICD-10 code I63.339) relies on a combination of clinical assessment, imaging studies, and laboratory evaluations. The presence of characteristic symptoms, supported by imaging findings of infarction in the posterior cerebral artery territory, is crucial for accurate diagnosis. Clinicians must also consider the patient's overall health and risk factors to guide treatment and management strategies effectively.

For further details on coding and clinical guidelines, healthcare professionals can refer to the ICD-10-CM coding manuals and relevant clinical practice guidelines[1][2].

Treatment Guidelines

Cerebral infarction due to thrombosis of the unspecified posterior cerebral artery, classified under ICD-10 code I63.339, represents a significant medical condition that requires prompt and effective treatment. This condition typically arises from a blockage in the blood supply to the brain, leading to tissue death and neurological deficits. Here, we will explore standard treatment approaches, including acute management, rehabilitation, and long-term care strategies.

Acute Management

1. Immediate Medical Intervention

  • Intravenous Thrombolysis: The primary treatment for acute ischemic stroke, including cerebral infarction due to thrombosis, is the administration of thrombolytic agents such as tissue plasminogen activator (tPA). This treatment is most effective when given within a 3 to 4.5-hour window from the onset of symptoms[1].
  • Endovascular Therapy: In cases where thrombolysis is not effective or feasible, mechanical thrombectomy may be performed. This procedure involves the physical removal of the clot using specialized devices, typically within 6 to 24 hours of symptom onset[2].

2. Supportive Care

  • Monitoring and Stabilization: Patients are closely monitored for vital signs, neurological status, and potential complications such as hemorrhagic transformation. Supportive care may include oxygen therapy, intravenous fluids, and management of blood pressure[3].
  • Antiplatelet Therapy: Following stabilization, antiplatelet medications such as aspirin or clopidogrel are often initiated to prevent further clot formation[4].

Rehabilitation

1. Physical Therapy

  • Rehabilitation is crucial for recovery post-stroke. Physical therapy focuses on improving mobility, strength, and coordination. Tailored exercise programs help patients regain independence and functionality[5].

2. Occupational Therapy

  • Occupational therapy assists patients in relearning daily activities and improving fine motor skills. This therapy is essential for enhancing the quality of life and promoting self-sufficiency[6].

3. Speech and Language Therapy

  • If the stroke affects speech or swallowing, speech-language pathologists provide targeted therapy to help patients regain communication skills and manage dysphagia[7].

Long-term Management

1. Secondary Prevention

  • Lifestyle Modifications: Patients are encouraged to adopt heart-healthy lifestyle changes, including a balanced diet, regular physical activity, smoking cessation, and weight management to reduce the risk of recurrent strokes[8].
  • Medications: Long-term antiplatelet therapy is often prescribed, and in some cases, anticoagulants may be indicated, especially if there are underlying conditions such as atrial fibrillation[9].

2. Regular Follow-up

  • Continuous follow-up with healthcare providers is essential for monitoring recovery progress, managing risk factors, and adjusting treatment plans as necessary. This may include regular imaging studies to assess cerebral blood flow and detect any new vascular issues[10].

Conclusion

The management of cerebral infarction due to thrombosis of the posterior cerebral artery involves a multifaceted approach that includes acute medical interventions, rehabilitation therapies, and long-term preventive strategies. Early recognition and treatment are critical to improving outcomes and minimizing long-term disabilities. Patients and caregivers should work closely with healthcare professionals to develop a comprehensive care plan tailored to individual needs and circumstances.

Related Information

Description

Clinical Information

  • Cerebral infarction due to PCA thrombosis
  • Blood flow interruption leads to tissue damage
  • Thrombosis in posterior cerebral artery (PCA)
  • Supplies blood to occipital and temporal lobes
  • Visual disturbances common
  • Homonymous hemianopia may occur
  • Cognitive impairments possible
  • Sensory loss, motor deficits, speech difficulties may arise
  • Ataxia, dysphagia, altered consciousness possible

Approximate Synonyms

  • Cerebral Infarction
  • Ischemic Stroke
  • Thrombotic Stroke
  • Posterior Cerebral Artery Infarction
  • Cerebral Thrombosis
  • Stroke
  • Neurological Deficit
  • Transient Ischemic Attack (TIA)
  • Vascular Occlusion
  • Cerebrovascular Accident (CVA)

Diagnostic Criteria

  • Sudden onset of neurological deficits
  • Visual disturbances or sensory loss
  • Motor weakness in affected areas
  • Homonymous hemianopia
  • Memory deficits and spatial awareness difficulties
  • Loss of gray-white matter differentiation on CT
  • Subtle hypodensities in affected area on CT
  • Restricted diffusion on DWI MRI
  • Thrombosis confirmed by angiography

Treatment Guidelines

  • Intravenous Thrombolysis within 3-4.5 hours
  • Endovascular Therapy within 6-24 hours
  • Monitoring and Stabilization with oxygen therapy
  • Antiplatelet Therapy to prevent further clot formation
  • Physical Therapy for mobility, strength, and coordination
  • Occupational Therapy for daily activities and fine motor skills
  • Speech and Language Therapy for communication and swallowing
  • Lifestyle Modifications for heart-healthy lifestyle changes
  • Medications including antiplatelets and anticoagulants
  • Regular Follow-up with healthcare providers

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